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PETS     

PETS - About your furry friends

Animals produce different types of substances that are the primary culprits in causing allergy. In general, proteins in the urine, saliva or dander (dead skin flakes) of pets provoke immune reactions in people with allergies. In cats, for example, the main substances that cause problems are proteins found in cat saliva, which get mixed up in house dust. In rodents such as mice or rats or guinea pigs, it seems that the urine contains the predominant substances that cause allergy.

DANDER

The problem is not with the pet's hair. It is the dander, or microscopic scales of dead skin (similar to, but much smaller than the dandruff on the human scalp) which pets are constantly shedding that is the cause. They are so tiny that you seldom, if ever, know that they are circulating in the air or laying on the furniture and carpets.

URINE

Recent studies indicate that urine (protein), from cats and dogs as well as from ferrets, guinea pigs, hamsters, and mice, contain allergens which can also trigger allergic reactions.

Biological contaminants can come from both outside and inside the home. Sources include house dust mites, animal dander, dust, pollen, cockroaches and molds, all of which can cause an allergic reaction. Some organisms can contaminate water sources, and become airborne through humidifiers

  • By-products of the building's heating, ventilation and
    air conditioning (HVAC) system, cooking and cigarette smoke.

  • Vapors "outgassing" from synthetic building materials,
    glues, carpets, furnishings and decorating materials.

  • Vapors from perfumes and some household cleaners.

  • The occupants: people and pets breathe out carbon dioxide
    and pets produce animal dander.

  • Outdoor air pollution entering into the home.

Toxoplasmosis (Toxoplasma gondii)

Toxoplasma gondii is found throughout North America and can infect almost any warm-blooded animal or bird, and humans. Infection with T. gondii, a condition called toxoplasmosis, can be very serious in humans. T. gondii can be passed from a pregnant woman to her fetus and cause abortions and congenital defects. In children and adults it can cause other signs, and is sometimes fatal. It can cause severe disease in persons with poor immune systems such as those undergoing chemotherapy or infected with human immunodeficiency virus (the virus that causes AIDS). The cases of toxoplasmosis in the United States had been declining to 350 cases annually in the early 1990’s. With the AIDS epidemic, the number of cases has increased. Surveys in the United States suggest 30% of cats and 25-50% of people have been infected (most clear the infection with no or few symptoms).

What are the signs and symptoms of toxoplasmosis in humans?

Humans can become infected either in utero, or by accidental ingestion of the oocysts. If T. gondii passes through the uterus of an infected pregnant woman to her fetus early in the pregnancy, spontaneous abortion is common. If the infection occurs later in pregnancy (10-24 weeks of gestation) the infant may have serious or fatal congenital defects including hydrocephalus, blindness, and mental retardation. Most infected pregnant women do not have symptoms. Approximately 60% of infected pregnant women will pass the infection to their fetus.

People infected through ingestion of oocysts may be listless and have fever, enlarged lymph nodes and less commonly, inflammation of the heart.

Cockroaches and flies may serve as transport hosts for T. gondii, carrying cat’s fecal material on their bodies. Controlling these pests may help in preventing the spread of T. gondii.

Diagnosis is generally made through serologic (blood) testing.

References

James T. Li, M.D., a Mayo Clinic board-certified allergist and internist

Dubey, JP. Toxoplasmosis. Zoonosis Updates. American Veterinary Medical Association, 1995:144-149.

Georgi, JR; Georgi, ME. Canine Clinical Parasitology. Lea & Febiger, Philadelphia PA; 1992;81,87-88.

Griffiths, HJ. A Handbook of Veterinary Parasitology. University of Minnesota Press, Minneapolis MN; 1978;31-33.

Hendrix, CM. Diagnostic Veterinary Parasitology. Mosby, Inc., St. Louis MO; 1998;22-23, 279-280.

Lappin, MR. Toxoplasmosis. Perspectives 1993(Charter Issue):8-16.

Lappin, MR. Immunodiagnosis and Management of Clinical Feline Toxoplasmosis. Presented at the Wisconsin Veterinary Medical Association Convention, 1994.

Lindsay, DS; Blagburn, BL; Dubey, JP. Feline toxoplasmosis and the importance of the Toxoplasma gondii oocyst. Compendium on Continuing Education for the Practicing Veterinarian, 1997; 19(4):448-461.

Sherding, RG. Toxoplasmosis, Neosporosis, and Other Multisystemic Protozoal Infections. In Birchard, SJ; Sherding, RG (eds): Saunders Manual of Small Animal Practice. WB Saunders Co., Philadelphia PA; 1994;141-145.

Sousby, EJL. Helminths, arthropods and protozoa of domesticated animals. Lea & Febiger, Philadelphia PA; 1982;670-682.

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